This is a 12 unit course to diagnosis of skin conditions related to TEXTURE. The surface texture and appearance of the epidermis is the first to show change when something goes wrong with skin and related systems, and this is what the client notices first. These changes are referred to as "site of response" and this is why all of the skin conditions related to TEXTURE will be our first choice of discussion.
- ► Introduction to the cells of the epidermis and dermis
- ► Understanding the importance of the lifecycle of the keratinocyte
- ► Diagnosing hyperkeratinisation
- ► Non Inflammatory Acne: Open & closed comedones & Inflammatory Acne grade three
- ► The lifecycle of the fibroblast, and how it is responsible for the formation of the connective tissue fibrils, collagen and elastin
- ► An in-depth look at the elastin fibril; from formation to ultimate deterioration
Hyperkeratinisation, Non inflammatory acne (grades 1-3) and Wound Healing
1. Introduction to the cells of the epidermis and dermis
This unit of learning consists of a brief refresher of the Pastiche Method of analysis followed by an introduction to all relevant cells of the epidermis and dermis, covering the key points of each cell and their respective roles. If it has been some time since you completed module one, this unit of learning will reacquaint you with the process and bring everything back to the forefront of your mind ready to more readily absorb the following units.
2. Understanding the importance of the lifecycle of the keratinocyte
We touched briefly on the epidermal and dermal cells responsible for each diagnostic characteristic in module one. For Texture, it was the keratinocyte and the fibroblast. The first epidermal skin condition under the diagnostic characteristic of Texture is hyper-keratinisation. Therefore, we begin our journey by first understanding this major cell of the epidermis, the keratinocyte, and the role it plays in creating the first lines of skin barrier defense.
3. Diagnosing hyperkeratinisation
In this unit we will be covering all the diagnostic clues that will pertain to hyperkeratinisation, exploring the common examples of leading causes of hyperkeratinisation; and linking these across to cells and systems affected. We examine the relationship between fluid intake, medications and diet in addition to the basic chemistry of cell requirements to function correctly. We then review how these causes manifest themselves on the skin surface.
4. Practical assignment in diagnosing hyperkeratinisation
In this unit we undertake out first practical assignment. Here we learn the basics of gathering the information from the consultation and relating it to the observations; then logically reaching a conclusion from the wide array of information. This process teaches you how to think three dimensionally and recognise the causative agents to the skin conditions. We will repeat this process across each of the categories of skin conditions as we move forward.
5. Non Inflammatory Acne: Open & closed comedones & Inflammatory Acne grade three
In this unit we examine the relationship of hormones, enzymes and amino acids to acne. We will discover how the health of the acid mantle and the skin’s flora play a role in aggravating the acne condition and how hyper keratinisation plays a key role. We also review the common extrinsic causative agents across diet, medication and topically applied substances, and how to properly grade the first three acne conditions.
6. The stages of the wound healing process
Before we move on to the units of learning covering skin conditions related to collagen and elastin, we review the wound healing process; and relate it to the various modalities used for treatments. This unit will help us to understand all that is known about the wound healing processes of skin; so as to apply that knowledge to the action of non ablative modalities commonly used in clinical services.
7. Loss of structural integrity, adhesion & resiliency and glycation: The fibroblast lifecycle
The fibroblast is the cell responsible for the formation of the connective tissue fibrils, collagen and elastin, including the dermal hydration fluid, the glycosaminoglycans. Changes to the supporting connective tissue of the dermis will accelerate the ageing appearance of skin. Because the ageing skin is a multifaceted skin condition, the skin diagnostic technician of today must have a more extensive base knowledge of the complete eco system that makes up the dermal structures.
8. Loss of structural integrity, adhesion & resiliency and glycation; Collagen fibril formation
Collagen fibres are a major component of the connective tissue of the dermis, tendons and ligaments. Consisting of closely packed fibrils with the diameter of the fibre dependent on the number of fibrils it contains. Learning about the seven different types of collagen that deteriorate during the ageing process and the progression of ageing through the dermis is an important part of the skin treatment therapist’s base knowledge. Learning to visually see this ageing pathway via histology slides and physical analysis is part of this segment of training.
9. Loss of structural integrity, adhesion & resiliency and glycation: Elastin fibril formation
Just as you learnt about the importance of the collagen fibril, this segment of your training program covers the elastin fibril, its formation and ultimate deterioration. Once again learning to visually see this deterioration via histology slides and physical analysis, this unit of learning completes another part of the puzzle in skin analysis.
10. Loss of structural integrity, adhesion & resiliency and glycation: Structure of the dermis
It has been established that the fibroblast is a major player in the creation of all dermal layers, and in the structural integrity, and density of skin. The dermis is comprised of seven layers, each one playing a special role in maintaining the shape and form of the face and body. The slow deterioration of these layers will be reflected in the outward appearance of skin. “A site of response”. Learning to determine the depth of ageing through the dermal structure will help you in your physical analysis of skin.
11. Loss of structural integrity, adhesion & resiliency and glycation: The physical analysis of the ageing skin
When connective tissue deterioration begins to compound, it will follow the easiest pathway away from the papillary and into the fascia septa. Being able to see this advancing ageing pathway by visual observation is very important during the physical skin analysis, it will assist your determining what treatment will be effective, how many, and how frequently. Using examples you will complete this segment of training with greater confidence in you diagnostic ability.
12. Loss of structural integrity, adhesion & resiliency and glycation: The practical assignment
In this unit we undertake practical assignment. Following a virtual client through from the consultation process using diagnostic forms and flow charts so as to logically reach a conclusion from the wide array of information gathered. This process teaches you how to think three dimensionally and recognize the causative agents to the skin conditions.